Human herpesvirus infections pathogenesis, diagnosis, and treatment

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Published by Raven Press in New York .

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Edition Notes

Book details

Statementeditors, Carlos Lopez, Bernard Roizman.
ContributionsLopez, Carlos, 1942-, Roizman, Bernard, 1929-, International Conference on Immunobiology and Prophylaxis of Human Herpesvirus Infections (2nd : 1985 : Fort Lauderdale, Fla.)
LC ClassificationsRC147.H6 H86 1986
The Physical Object
Paginationxiv, 304 p. :
Number of Pages304
ID Numbers
Open LibraryOL2724254M
ISBN 100881672351
LC Control Number86017908

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1. Introductory Chapter: Human Herpesvirus - A Short Introduction. By Ronaldo Luis Thomasini. Open access peer-reviewed.

Sunlight and Herpes Virus. By Vittorio Mazzarello, Marco Ferrari, Stefano Decandia and Maria Alessandra Sotgiu. Open access peer-reviewed.

Neurologic Complications of Varicella-Zoster Virus InfectionAuthor: Ronaldo Luis Thomasini. The Herpesviruses: Immunobiology and Prophylaxis of Human Herpesvirus Infections (The Viruses) Softcover reprint of the original 1st ed. Edition by Carlos Lopez (Editor), Bernard Roizman (Editor).

One of the most important features of the book is that it covers aspects of both basic research and clinical medicine. Herpesviridae, a family of double-strand DNA viruses, has unique biological features by which these viruses establish latency after primary infection and reactivate in later life.

Exanthem Subitum and Human Herpesvirus 6 (HHV-6) Infection. Koichi Yamanishi, Toshio Kondo, Kazukiro Kondo, Yasuhiko Hayakawa, Shinichi Kido, Kazuo Takahashi et al. Pages Viral Pathology of Human Herpesvirus 6 Infection.

ISBN: OCLC Number: Description: ix, pages: illustrations ; 24 cm. Contents: Herpes simplex virus infections / Ervin Adam --Human cytomegalovirus infections / John L. Sullivan and James B. Hanshaw --Varicella-zoster virus infections / Charles Grose --Epstein-Barr virus and infectious monoucleosis / Werner Henle and Gertrude Henle --Association of Epstein.

Red Book: ; Red Book: Errata; Red Book: ; Red Book: Errata; Red Book: ; Infection Control and Prevention for Hospitalized Children Human Herpesvirus 8 p Human Immunodeficiency Virus Infection p I.

Human herpesviruses type 6 (HHV-6) and type 7 (HHV-7) are ubiquitous, lymphotropic, β-herpesviruses. 1, 2 They are 2 of the known causative agents of roseola infantum, also known as exanthem subitum.

3, 4 It is now understood that in otherwise healthy children, the spectrum of diseases associated with HHV-6 and HHV-7 is far broader than the benign illness of roseola.

Epstein–Barr virus infection after adolescence and age independent human herpesvirus 6A infection are risk factors for multiple sclerosis. Clinical manifestations of primary infection with human herpesvirus 6B (HHV-6B) include roseola (exanthem subitum) in approximately 20% of infected children, as well as a nonspecific febrile illness without rash or localizing signs.

Acute HHV-6B infection may be accompanied by cervical and characteristic postoccipital lymphadenopathy, gastrointestinal tract or respiratory tract signs, and. Overview of Herpesvirus Infections By. Kenneth M. Kaye, MD, Brigham and Women’s Hospital, Harvard Medical School (EBV) and human herpesvirus type 8 (HHV-8), also known as Kaposi sarcoma–associated herpesvirus (KSHV), can cause certain cancers.

Roseola infantum is a childhood disease caused by herpesvirus 6 (and sometimes 7). Herpesvirus group can establish primary infections with nonsevere symptoms, which can result in an efficient immune response that prevents a new infection.

However, the virus is not completely eliminated, its genome remains within cells without productive : Ronaldo Luis Thomasini.

Thomas M. Donnelly BVSc, Diplomate ACLAM, in Ferrets, Rabbits, and Rodents (Second Edition), Viral Infections. Chinchillas are susceptible to human herpesvirus 1 and may play a role as a temporary reservoir for human infections.

Goudas and Giltoy 22 described a spontaneous, herpeslike viral infection in a female chinchilla. Wohlsein et al. 95 reported a 1-year-old male chinchilla with a.

There is no loftier aim than to cure or prevent human infections with these viruses. The objective of much of the current research on herpesviruses is directed toward an understanding of the molecular mechanisms involved in initiation of infection, establish­ ment and termination of latent state, virus multiplication, and the destruction of.

The spectrum of viruses known to cause encephalitis is continuously growing. Lately, human herpesvirus 6 variants A and B (HHV-6A and HHV-6B) (Salahuddin et al.

) and HHV-7 (Frenkel et al. ) (Herpesviridae family, Betaherpesvirinae subfamily, Roseolovirus genus) have been identied. Interstitial pneumonitis associated with human herpesvirus-6 infection after marrow transplantation.

Lancet. Jul 20; ()– Cone RW, Hackman RC, Huang ML, Bowden RA, Meyers JD, Metcalf M, Zeh J, Ashley R, Corey L. Human herpesvirus 6 in lung tissue from patients with pneumonitis after bone marrow transplantation.

The present book presents a comprehensive review of Human Herpesvirus-6 (HHV-6) infects up to 90% of the world’s population and can cause potentially life-threatening diseases. Clinicians typically. Striking changes have occurred in the world since the publication of the last edition of Viral Infections of Humans.

The global population is rapidly approaching 8 billion; climate change is leading to the introduction of new hosts, vectors and virus diseases heretofore never seen in many parts of the world; technological advances have revolutionized the ability to recognize and characterize.

Electron microscopy revealed a novel virus that they named Human B-Lymphotrophic Virus (HBLV). Shortly after its discovery, Ablashi et al. described five cell lines that can be infected by the newly discovered HBLV.

They published that HSB-2, a particular T-cell line, is highly susceptible to infection. Human herpesvirus 6 (HHV-6) and human herpesvirus 7 (HHV-7) (HPV) infection Human parainfluenza viruses (HPIV) Human parainfluenza virus infection Hymenolepis nana and Hymenolepis diminuta: Hymenolepiasis: Red Book: Report of the Committee on Infectious Diseases.

Human herpesvirus 6 (HHV-6) is a lymphotropic virus that belongs to the β-herpesviruses, together with its close relatives cytomegalovirus (CMV) and human herpesvirus 7. In infants, HHV-6 infection may present either as excanthema subitum or as nonspecific infection, or.

Human herpesvirus 6(HHV-6) is a human pathogen of emerging clinical significance. HHV-6 was first isolated from patients with lymphoproliferative disorders in (Salahuddin et al., ).HHV-6 isolates are classified into two groups as variants A(HHV-6A) and variant B(HHV-6B) (Schirmer et al., The two variants are closely related but show consistent differences in biological.

Get this from a library. Immunobiology and prophylaxis of human herpesvirus infections. [Carlos Lopez;] -- The proceedings of the October Conference deal with the new viruses and the molecular basis of diseases caused by viruses that have been known for.

Purchase Human Herpesvirus-6, Volume 4 - 1st Edition. Print Book & E-Book. ISBNBook Edition: 1. Human herpesvirus 6 (HHV-6) is a member of the Herpesviridae family. Like other members of this family, the virus remains in a latent state after primary infection has resolved and can reactivate. HHV-6 encephalomyelitis is an uncommon clinical manifestation in immuncompetent adults.

Kaposi's sarcoma-associated herpesvirus (KSHV) is the ninth known human herpesvirus; its formal name according to the International Committee on Taxonomy of Viruses (ICTV) is Human gammaherpesvirus 8, or HHV-8 in short.

Like other herpesviruses, its informal names are used interchangeably with its formal ICTV name. This virus causes Kaposi's sarcoma, a cancer commonly. Joseph Ongrádi, Dharam V. Ablashi, in Human Herpesviruses HHV-6A, HHV-6B & HHV-7 (Third Edition), Human Herpesvirus 7. Human herpesvirus 7 was isolated from activated T lymphocytes of a healthy donor.

8 HHV-7 is shed in the saliva of healthy subjects. It establishes latent infection in CD4+lymphocytes and salivary glands.

A prospective case control study of the association of Gianotti-Crosti syndrome with human herpesvirus 6 and human herpesvirus 7 infections. Pediatr Dermatol ; Boccara O, Lesage F, Regnault V, et al.

Nonbacterial purpura fulminans and severe autoimmune acquired protein S deficiency associated with human herpesvirus-6 active replication.

Like all strains of human herpesvirus, HHV-6 causes a primary infection, such as roseola infantum, and then stays latent in the body, where it can be reactivated by stress or other illness.

In some adults who experience a primary infection of HHV-6, a. Human herpesvirus 6 (HHV6) may be an important pathogen following allogeneic hematopoietic cell transplantation (HCT). We prospectively evaluated weekly HHV6 viremia testing after allogeneic HCT using a quantitative polymerase chain reaction (PCR)-based assay.

HHV-6. There are eight herpesviruses for which humans are the primary host. They are the herpes simplex virus 1, herpes simplex virus 2, varicella-zoster virus, Epstein-Barr virus, cytomegalovirus, Human herpesvirus-6, Human herpesvirus-7, and Kaposi’s sarcoma herpes virus.

Epidemiology/ Geographic distribution. Human herpesvirus 6 (HHV-6), a member of the beta-herpesvirinae subfamily, is highly seroprevalent, has a worldwide distribution, and infection usually occurs within the first two years of life.

In this age group, HHV-6 causes febrile illness including exanthem subitum with seizures a recognised com. Infections with human herpesvirus 6 (HHV-6), a [Beta]-herpesvirus of which two variant groups (A and B) are recognized, is very common, approaching % in seroprevalence.

Primary infection with HHV-6B causes roseola infantum or exanthem subitum, a common childhood disease that resolves spontaneously.

Human herpesvirus 6 (HHV-6) and Human herpesvirus 7 (HHV-7) Fasciolopsiasis: Fasciolopsis buski: Human bocavirus infection Human bocavirus (HBoV) Human ewingii ehrlichiosis: Ehrlichia ewingii: Human granulocytic anaplasmosis (HGA) Red Book: Report of the.

Organized into 21 chapters, this book starts with an overview of the classification of the herpesvirus and proceeds to explore the origins and phylogeny of the herpesviruses. This text then examines the earliest electron microscopic studies on the morphology of the herpesviruses by using shadowcast preparations of herpes simplex virus and of.

Human Herpesvirus 7 HHV Infour years after the isolation of HHV-6, another herpesvirus was discovered. Because of the similarities in genes HHV-6 and HHV-7 and the fact that HHV-7 often acts together with HHV-6, and the viruses together are sometimes referred to as Roseolovirus.

HIV Serodiscordant Sex Partners and the Prevalence of Human Herpesvirus 8 Infection among HIV Negative Men Who Have Sex with Men: Baseline Data from the EXPLORE Study Corey Casper, D. Carrell, K. Miller, F. Judson, A. Meier, John S.

Pauk, R. Morrow, Lawrence Corey, Anna Wald and Connie L. Celum, Sexually Transmitted Infections,   Hall CB, Caserta MT, Schnabel K, Shelley LM, Marino AS, Carnahan JA, et al.

Chromosomal integration of human herpesvirus 6 is the major mode of congenital human herpesvirus 6 infection. Pediatrics. ;(3)– pmid. Herpes simplex virus is common in the United States. There are two types of the virus, HSV-1 and HSV HSV-1 is known as oral herpes, and HSV-2.

Abstract. BackgroundLittle is known about the clinical and virologic manifestations of human herpesvirus (HHV)–8 infection in immunocompetent persons in the absence of malignancy. MethodsA total of 46 human immunodeficiency virus–negative, HHV-8–seropositive men collected saliva daily, and 25 recorded 15 common symptoms daily (gastrointestinal, constitutional, and.

Human herpesvirus infections are endemic and sexual contact is a significant method of transmission for several including both herpes simplex virus 1 and 2 (HSV-1, HSV-2), also human cytomegalovirus (HHV-5) and likely Karposi's sarcoma herpesvirus (HHV-8). The increasing prevalence of genetial herpes and corresponding rise of neonatal infection.

The seroprevalence of human herpesvirus-8 (HHV-8)—also known as Kaposi sarcoma-associated herpesvirus (KSHV)—varies worldwide and is estimated to be 1% to 5% in the general U.S. population 1,2 compared with 10% to 20% in certain Mediterranean countries and 30% to 80% in parts of sub-Saharan Africa.

3 In the United States, men who have sex. Human herpesvirus 6 (HHV-6) was the sixth herpesvirus discovered. Isolated in during attempts to find novel viruses in patients with lymphoproliferative diseases, HHV-6 is now recognized as a T-cell lymphotropic virus with high affinity for CD4 lymphocytes.Background Infection with human herpesvirus-6 (HHV-6) is nearly universal in infancy or early childhood.

However, the course of this infection, its complications, and its potential for persistence.

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